Tomorrow is the first day of school for a lot of medical, dental, nursing, physician assistant, medical technology, veterinary, and other allied health students. I hope I didn’t miss anyone.
Q. I can’t seem to get the different types of necrosis straight (liquefactive, fibrinoid, etc.). Any help?
Sometimes in pathology, you hear the name of a disease over and over, but somehow you never really learn about it.
Here’s a long term: leukoerythroblastotic reaction. Despite its length, it’s a pretty good term, because it describes a reactive condition in which you see young red cells (erythroblasts) and young white cells (leuko-) out in the peripheral blood.
One thing that’s hard to get the hang of in pathology is the difference in appearance between chronic and acute inflammation in tissue sections.
We talk a lot about strokes in a clinical way in medical school. We discuss which areas of the brain are involved, and we correlate the areas damaged with the patient’s symptoms.
Q. Why is there a microangiopathic hemolytic anemia in thrombotic thrombocytopenic purpura and disseminated intravascular coagulation?
Cytokines are little messenger molecules that are involved in tons of different physiologic and pathologic conditions.
Q. I have a question. Why do you see a decreased glomerular filtration rate in nephritic syndrome? I read on your blog and other places that it’s due to “hemodynamic changes”– from Robbins I’m assuming this is compensatory stuff- but wouldn’t that increase GFR? A. It’s because of what’s going on in the glomerulus! In [...]
